Research Summary
The Iritani lab is interested in understanding the molecular mechanisms that control the development and functions of lymphocytes, and how when dysregulated, these signaling pathways can result in immunodeficiency diseases, autoimmunity, and/or cancer. Utilizing a chemical mutagenesis screen in mice, we identified several novel signaling molecules that are essential for lymphocyte development by regulating cell metabolism and the actin cytoskeleton respectively. We utilize transcriptomics, proteomics, and metabolomics in murine models for understanding how these molecules function.
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Publications
The following publications were retrieved from PubMed:
Folliculin Interacting Protein 1 Maintains Metabolic Homeostasis during B Cell Development by Modulating AMPK, mTORC1, and TFE3.
Ramírez JA, Iwata T, Park H, Tsang M, Kang J, Cui K, Kwong W, James RG, Baba M, Schmidt LS, Iritani BM.
J Immunol. 2019 Dec 1; 11(203)2899-2908
Loss of Fnip1 alters kidney developmental transcriptional program and synergizes with TSC1 loss to promote mTORC1 activation and renal cyst formation.
Centini R, Tsang M, Iwata T, Park H, Delrow J, Margineantu D, Iritani BM, Gu H, Liggitt HD, Kang J, Kang L, Hockenbery DM, Raftery D, Iritani BM.
PLoS One. 2018; 6(13)e0197973